Synopsis

Source of validity: Scientific
Rating (out of 5): 5

Vitamin K, including its form menaquinone (vitamin K2), is scientifically validated for the treatment and prevention of bleeding, particularly when resulting from vitamin K deficiency. Vitamin K plays a crucial role in the synthesis of clotting factors II, VII, IX, and X in the liver. Without adequate vitamin K, these clotting factors are produced in an inactive form, leading to impaired coagulation and an increased risk of bleeding, both internal and external.

Clinically, vitamin K is routinely administered to treat or prevent bleeding disorders caused by deficiency, such as hemorrhagic disease of the newborn, over-anticoagulation with warfarin, or certain malabsorption syndromes. Its use in these contexts is standard medical practice, supported by decades of clinical research and consensus guidelines. For example, vitamin K1 (phytonadione) is the most commonly used form in acute care, but menaquinones also contribute to vitamin K status through dietary and supplemental sources. Administration of vitamin K rapidly restores the synthesis of active clotting factors, effectively halting bleeding episodes due to deficiency.

However, for bleeding not caused by vitamin K deficiency (such as trauma or inherited hemophilia), vitamin K supplementation is not effective. In summary, vitamin K’s use for treating or preventing bleeding is strongly evidence-based, but is specifically effective when bleeding is linked to a deficiency in vitamin K–dependent clotting factor production.

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