Vitamin B12 (cobalamin) is strongly supported by scientific evidence as an essential nutrient for the health of the nervous system. It plays a crucial role in the synthesis and maintenance of myelin, the protective sheath around nerves, and is involved in the production of neurotransmitters and DNA synthesis. Deficiency of vitamin B12 leads to neurological symptoms such as numbness, tingling, balance problems, cognitive disturbances, and, in severe cases, irreversible nerve damage. The connection between B12 deficiency and neurological dysfunction is well established in medical literature, with numerous clinical studies showing improvement or stabilization of nerve-related symptoms following B12 supplementation in deficient individuals.

B12 deficiency is especially relevant in populations with poor dietary intake (such as vegetarians and vegans), malabsorption syndromes, or certain genetic conditions. The scientific validation for B12’s role in supporting the nervous system is reflected in standard clinical practice: B12 supplementation is recommended to prevent and treat neurological complications caused by deficiency. It is not merely traditional use but rather a well-characterized biochemical necessity, with strong clinical evidence supporting its critical importance for nerve health.

References: - O’Leary, F., & Samman, S. (2010). Vitamin B12 in health and disease. Nutrients, 2(3), 299-316. - Hunt, A., Harrington, D., & Robinson, S. (2014). Vitamin B12 deficiency. BMJ, 349, g5226.