IngredientsCoffee fruit — also called the coffee cherry, coffee berry, or cascara (from the Spanish cáscara, meaning "husk" or "skin") — is the mature drupe produced by plants of the genus Coffea, family Rubiaceae. Coffee cherry tea is an herbal preparation made from the dried skins and/or pulp of the fruit of the coffee plant that remain after the coffee beans have been collected from within; it is also known as cascara, from the Spanish cáscara, meaning "husk." The term "cascara" in this context must be distinguished from cascara sagrada, an unrelated laxative derived from Rhamnus purshiana.
The principal commercially cultivated species are Coffea arabica, Coffea robusta (C. canephora), and Coffea liberica. Globally, the majority of commercial coffee bean production is derived from Coffea arabica L. (57%) and Coffea canephora Pierre ex A. Froehner (42%). The coffee fruit of Coffea arabica is the source most commonly exploited for supplement products, cascara beverages, and whole-fruit extracts.
C. arabica's origin is estimated to have occurred approximately 50,000 years ago in the highlands (between 1,600 and 2,800 m) of the tropical rainforests of southeastern Ethiopia, the Boma Plateau in Sudan, and on Mount Marsabit in Kenya. The wild coffee plant is native to Ethiopia, where it was discovered approximately 850 AD and subsequently cultivated in the Arab colony of Harar. Today, coffee fruit is harvested across the tropical "bean belt," encompassing Latin America, sub-Saharan Africa, and Southeast Asia.
A coffee cherry is a drupe — the same structural category as a cherry, plum, or olive. Inside the fruit, two coffee seeds sit surrounded by pulp, a sticky layer called mucilage, and a tough inner parchment. More precisely, the pericarp comprises three outer layers of the fruit: the exocarp (skin), the mesocarp (mucilage), and the endocarp (parchment). The coffee seed or bean comprises a silver skin, endosperm, and an embryo.
The material exploited for cascara and related products comprises the skin (exocarp), pulp (mesocarp), mucilage (pectin), parchment (endocarp), and a portion of the silver skin of the coffee fruit, and consists mainly of digestible carbohydrates, dietary fibre, and water. The mature coffee fruit comprises: fruit skin, which accounts for an estimated 29–43% (w/w); endocarp (parchment), which constitutes approximately 12% (w/w); silverskin, which accounts for around 4.2% (w/w); and coffee beans, which comprise approximately 55% (w/w) of the fruit weight.
The non-seed portions of the coffee fruit are processed and consumed in several distinct forms:
The Arabic word for the coffee cherry husk is "qishr," and the Yemeni tradition of brewing it is documented as far back as the 15th century. This predates by several decades the earliest clear evidence of roasted coffee bean brewing, which appears to have emerged in Sufi monasteries in Yemen around the same period. In Yemen, a similar drink called qishr was developed around the same time. This traditional Yemeni beverage is made with ginger, cinnamon, and other spices, and remains widely popular in Yemen to this day, even more so than coffee itself. It is believed that cáscara tea was consumed in Yemen even before the form of coffee we know today.
In Ethiopia, a similar beverage called hashara is consumed in coffee-growing regions. The historical record suggests these drinks may predate the roasting and grinding of coffee beans as a beverage practice — the seed that became the global commodity may have been discovered because someone was already familiar with the plant's fruit. Since coffee cultivation is thought to have originated in Ethiopia as early as the 9th century, it is reasonable to assume that Ethiopia's version of coffee cherry tea — called hashara or geshar — has existed just as long. To prepare this warm drink, coffee husks are toasted and steeped generously for a strong, full-bodied cup.
From geshar and qishr in Ethiopia and Yemen, to cascara in Latin America — or sultana in Bolivia — coffee cherry tea has quietly existed in the wings for centuries. However, the primary tradition firmly documented in historical sources is concentrated in the Horn of Africa and the Arabian Peninsula. There is not a history of widespread use of coffee skin as tea in Latin America that can be historically verified. There is an 800+ year old tradition of coffee fruit skin tea in the horn of Africa and Yemen.
Starting about 2005, cascara was independently developed and promoted for export by Salvadoran coffee farmer Aida Batlle. Around 2005 she noticed the coffee cherry husks, which are commonly discarded in the milling process, had a floral aroma, and she decided to try brewing tea from them. By 2009 it was being offered as a beverage in US coffee shops. Increasing demand for cáscara from large U.S.-based coffee chains has, in some cases, led to the dried husks fetching higher prices than the coffee beans.
Outside of these traditional uses, in most coffee-producing countries the coffee fruit is usually considered a wasted byproduct of the coffee-production process.
Chlorogenic acids (CGAs) are the dominant bioactive polyphenols in coffee fruit. Chlorogenic and isochlorogenic acids are naturally occurring antioxidant dietary polyphenolic compounds found in high concentrations in plants, fruits, vegetables, coffee, and coffee by-products. An electronic literature search has revealed that 5-caffeoylquinic acid (5-CQA) and 3,5-dicaffeoylquinic acid (3,5-DCQA) are the major chlorogenic acids found in coffee by-products. Coffee cherry is rich in chlorogenic acids (notably 5-caffeoylquinic acid), caffeine, and polyphenols that inhibit α-glucosidase and α-amylase enzymes while elevating Brain-Derived Neurotrophic Factor (BDNF) to support cognitive and metabolic health.
The fruit's non-seed layers contain caffeine, though typically at lower concentrations than the roasted bean. Fresh coffee cherries contain 70–110 milligrams of caffeine per 100 grams of whole fruit without the seed. The EFSA scientific panel noted that considering 100% extraction of caffeine from the dried husk to the beverage, the maximum concentration of caffeine in infusions produced using the novel food could be up to 600 mg/L of drink, a concentration comparable to those in coffee beverages.
Coffee fruit (pulp and skin) contains anthocyanins, proanthocyanidins, and phenolic acids. Coffee pulp has four significant constituents: chlorogenic acid, caffeine, epicatechin, and catechin. Constitutive proanthocyanidin units in coffee pulp are mainly epicatechin, representing more than 90% of the proanthocyanidin units, with average degrees of polymerization in the range of 3.8–9.1. Monomer to hexamer units of flavan-3-ols from fresh coffee pulp have been separated by normal-phase HPLC. Additional phenolics include: caffeic, chlorogenic, p-coumaric, ferulic, and sinapic acids, rutin, quercetin, kaempferol, and isoquercitrin identified in fruits of C. arabica.
Diterpene-type substances such as kafestol and kahweol have been isolated, characterized, and chemically defined from Coffea arabica, with documented physiological effects on the human organism. Kahweol and cafestol have also been tentatively identified in coffee samples by LC-ESI-QTOF-MS/MS analysis.
The most abundant bioactive metabolites in coffee include caffeine, quinic acid derivatives (chlorogenic acid), caffeic acid, ferulic acid, p-coumaric acid, melatonin, and serotonin. Coffee pulp also contains rutin and epicatechin.
The primary molecular mechanisms proposed and, to varying degrees, studied include:
The most developed clinical research program on coffee fruit as an isolated supplement concerns its effects on cognitive function, primarily mediated through modulation of BDNF.
A single-dose study was performed to assess the effect of whole coffee fruit concentrate powder (WCFC), green coffee caffeine powder, grape seed extract powder, and green coffee bean extract powder on blood levels of BDNF. Randomly assorted groups of fasted subjects consumed a single 100 mg dose of each material. Plasma samples were collected at time zero (T0) and at 30-minute intervals, up to 120 minutes. Two control groups were included: subjects treated with silica dioxide (as placebo) or with no treatment. The collected data revealed that treatment with WCFC increased plasma BDNF by 143% (n=10) compared with baseline. These results indicate that WCFC could be used for modulation of BDNF-dependent health conditions; however, larger clinical studies are needed to support this possibility. This study, published in the British Journal of Nutrition, had a very small sample (n=10 per group) and used a within-subject pilot design — limitations that constrain interpretation.
A subsequent within-subject study involving 20 healthy adults (ages 25–35) confirmed this finding. All fasted and resting subjects received placebo on Day 1, WCFC on Day 2, and a cup of freshly brewed coffee on Day 3. Treatment with WCFC resulted in a statistically significant increase in plasma BDNF compared to placebo (p = 0.0073) or coffee (p = 0.0219) during the first 60 minutes. Oral WCFC consumption also acutely increased BDNF levels in serum exosomes. All presented results justify further clinical investigation of WCFC as a tool to manage BDNF-dependent health conditions.
A remote, randomized, double-blind, placebo-controlled clinical trial published in Nutrients (2024; doi: 10.3390/nu16142348) by Robinson et al. investigated the cognitive effects of whole coffee cherry extract. Participants were randomized into one of two intervention arms: placebo or 200 mg CCE. Acute results supported that CCE outperformed placebo, reducing omissions and improving accuracy during working memory and inhibitory control tasks. Long-term results also indicated that CCE outperformed placebo on a measure of accuracy. To be eligible, participants had to reside in the United States, be between 40 and 65 years of age, with no known psychiatric or neurological conditions, and be generally healthy. Notably, one of the study's authors (John M. Hunter) was affiliated with VDF FutureCeuticals, Inc., the commercial producer of the tested extract, which is a relevant conflict of interest to acknowledge when evaluating the strength of this evidence.
Preliminary and pilot studies have demonstrated that CCE may improve several measures of cognition in older adults (i.e., 55–65 years of age) exhibiting mild cognitive decline, and that these improvements may be driven by distinct neurophysiological changes. A registered trial (NCT03812744) investigated neurophysiological effects in older adults, proposing that increases in serum concentrations of both serum total and exosomal BDNF may represent one of the mechanisms responsible for improved cognitive function after acute WCCE administration.
Not all cognitive studies have been positive. A randomized, double-blind, placebo-controlled crossover study conducted at Northumbria University revealed no effect of 300 mg coffeeberry extract on cognition, while 100 mg resulted in increased mental fatigue during the performance of cognitively demanding tasks. The study aimed to replicate previous findings by investigating the effects of low (100 mg) and moderate (300 mg) doses of chlorogenic acid-rich coffeeberry extract on cognition and mood; it followed a randomized, double-blind, placebo-controlled crossover design with four study arms. Research took place from July 2021 to January 2022 at the Brain, Performance and Nutrition Research Centre (BPNRC) at Northumbria University. These null and negative findings underscore that the cognitive evidence base remains preliminary and inconsistent.
Evidence strength (cognitive): Preliminary to moderate. The BDNF elevation findings are replicated across at least two small human studies, and the 2024 RCT provides the strongest evidence to date for working memory improvements with 200 mg CCE. However, study sizes are small, trial durations are short, some studies have commercial conflicts of interest, and at least one independent crossover study failed to replicate positive cognitive effects. Larger independent trials are required.
A total of 61 participants were randomly divided into a study group (n=30), receiving coffee cherry pulp juice concentrate, and a control group (n=31), receiving a placebo drink of 14 g twice daily for 12 weeks. Adverse symptoms, body weight, hematological and biochemical parameters, vital signs, and heart function were evaluated. The results showed no intervention-related adverse events. Body weight, liver, renal function, complete blood counts, blood glucose, urinalysis, and electrocardiograms were not significantly altered throughout the study. Consuming the juice for at least 8 weeks significantly decreased cholesterol and LDL levels. The glucose levels were maintained significantly better than those of the placebo group. The findings suggest that continuously consuming 28 g/day of coffee pulp juice concentrate for 12 weeks is safe in healthy volunteers. This study (Rungraung N et al., 2023; PMID 37049443) represents the primary human evidence for lipid and glucose effects. Results regarding cholesterol and glucose maintenance are encouraging but come from a single, relatively small trial in healthy — not metabolically impaired — volunteers.
In vitro and in vivo studies in rats indicate that selected coffee extract may improve exaggerated postprandial spikes in blood glucose via inhibition of intestinal sucrase, thus delaying carbohydrate absorption. These in vitro and in vivo studies could provide the biochemical rationale for further clinical study of coffee-based dietary supplements.
Evidence on chlorogenic acid-rich green coffee bean extracts (GCE) — distinct from whole coffee fruit preparations but sharing the key CGA constituent — is relevant as a mechanistic analog. Chlorogenic acid seems to have hypoglycemic effects similar to metformin by improving insulin resistance, evidently without adverse effects; however, clinical trials on the glucose-lowering effects of GCE and CGA are controversial and contradictory. Several studies indicated that GCE supplementation is inversely related to hyperglycemia and insulin resistance, while some studies did not support these outcomes. Chronic CGA supplementation (4–12 weeks) has been linked to reduced body mass, waist circumference, fasting glucose, and insulin resistance in both healthy adults and those with metabolic disease. These GCE studies are not directly equivalent to whole coffee fruit studies and cannot be conflated, but they inform mechanistic plausibility.
Evidence strength (metabolic): Preliminary. The single human study using coffee cherry pulp juice concentrate (Rungraung et al., 2023) showed promising lipid and glucose effects in healthy volunteers. Broader evidence from GCE studies (sharing chlorogenic acid content) adds mechanistic plausibility, but dedicated large-scale RCTs in metabolically impaired populations using coffee fruit preparations are lacking.
Consistent studies have related the intake of dietary phenolic compounds with a decreased risk of chronic diseases associated with oxidative stress, such as cardiovascular diseases and cancer. Coffee pulp is a source of antioxidant phytochemicals, including phenolic compounds and caffeine. However, the antioxidant properties of the phenolic compounds from coffee pulp are physiologically limited by their bioaccessibility, bioavailability, and biotransformation occurring during gastrointestinal digestion. Toxicological, pharmacokinetic, and clinical data from animal and human studies indicated no significant evidence of toxic or adverse effects following acute oral exposure. The current state of knowledge suggests that long-term exposure to chlorogenic and isochlorogenic acids by daily consumption does not appear to pose a risk to human health.
In vitro research has demonstrated that purification of coffee pulp extract increases antioxidant activity substantially: antioxidant activity was greatly increased after the purification process. Compared to un-purified crude coffee extract, the antioxidant activity of the purified samples increased approximately 34%, assumed to occur due to the slight increase of chlorogenic acid and caffeic acid.
Evidence strength (antioxidant): Mechanistically well-established in vitro; the relationship between in vitro antioxidant metrics and meaningful clinical outcomes in humans has not been demonstrated in dedicated coffee fruit trials.
Coffee-derived materials have been reported to contain polyphenols, alkaloids, and flavonoids associated with antioxidative and anti-inflammatory activities across different plant parts. In vitro studies demonstrate antioxidant, anti-aging, anti-inflammatory, photoprotective, wound-healing, and antimicrobial activities. Animal models show photoprotection and wound-healing effects. Regarding topical use of coffee pulp extract specifically, one study assessed anti-microbial and wound-healing properties: the Robusta IAC extract had the greatest prominence with 192.92 μg/mL of chlorogenic acid and was not cytotoxic, with a MIC of 3 mg/mL against Staphylococcus aureus. This extract was incorporated into a stable formulation preferred by 88% of volunteers. A scratch assay exhibited that the formulation promoted cell migration after 24 h.
Evidence strength (dermatological): Preclinical (in vitro and animal). No rigorous human clinical trials on dermatological endpoints using coffee fruit–specific preparations have been identified in the peer-reviewed literature.
Coffee pulp extract (CPE) therapy suppressed the synthesis of inflammatory cytokines and mediators — including TNF-α, IL-6, IL-1β, COX-2, iNOS, and nitric oxide, as well as prostaglandin E2 — in LPS-activated murine macrophage cells. Studies have found that coffee polyphenols, such as chlorogenic acids, have many health-promoting properties, including antioxidant, anti-inflammatory, anti-cancer, anti-diabetes, and antihypertensive properties. These mechanistic findings are based on cell culture and animal models. Human interventional data on coffee fruit's anti-inflammatory endpoints is not yet available.
Evidence strength (anti-inflammatory): Preclinical only. No human trials specifically measuring inflammatory markers using coffee fruit preparations have been identified in the reviewed sources.
The following dosages are drawn directly from cited clinical studies:
No official recommended dietary allowance (RDA) or tolerable upper intake level (UL) has been established for any coffee fruit preparation by any regulatory body.
Following a request from the European Commission, the EFSA Panel on Nutrition, Novel Foods and Food Allergens (NDA) delivered an opinion on dried coffee husk (cascara) from Coffea arabica L. as a novel food pursuant to Regulation (EU) 2015/2283. The novel food comprises the skin (exocarp), pulp (mesocarp), mucilage (pectin), parchment (endocarp), and a portion of the silver skin of the coffee fruit, and consists mainly of digestible carbohydrates, dietary fibre, and water. The Panel considers that there are no safety concerns regarding the stability of the novel food if it complies with proposed specification limits during its entire shelf-life. The consumption of beverages containing caffeine is not recommended for children, pregnant or breast-feeding women if the caffeine content exceeds 150 mg/L. Taking into account the nature of the novel food, the history of use as food, and the proposed uses and use levels, the Panel considers that no toxicological studies are required on the novel food. The risk of allergic reactions is considered low. The Panel concludes that the dried husk of the fruit of Coffea arabica L. is safe under the proposed conditions of use.
The maximum concentration of caffeine in infusions produced using dried coffee husk could be up to 600 mg/L of drink, a concentration comparable to those in coffee beverages. Accordingly, cascara infusions may deliver substantial caffeine loads, particularly if prepared at high concentrations or consumed in large volumes. This is the EFSA panel's primary identified safety issue for the general population.
A 12-week clinical safety study (Rungraung N et al., 2023, PMID 37049443) found coffee cherry pulp juice concentrate to be well-tolerated in healthy adults with no serious adverse events, though mild gastrointestinal symptoms (bloating, mild nausea) were occasionally reported due to its fiber and caffeine content.
Because coffee fruit preparations contain caffeine, the same interaction considerations relevant to caffeine apply:
Toxicological, pharmacokinetic, and clinical data from animal and human studies indicated no significant evidence of toxic or adverse effects following acute oral exposure to chlorogenic and isochlorogenic acids. The current state of knowledge suggests that long-term exposure to these compounds by daily consumption does not appear to pose a risk to human health.
Coffee cherry skin and pulp products are considered a novel food according to the European Union's and Thailand's Novel Food legal regulations. In the United States, whole coffee fruit products are generally marketed as dietary supplements under the Dietary Supplement Health and Education Act (DSHEA); the FDA has not issued a specific regulatory determination on whole coffee fruit preparations equivalent to EFSA's novel food opinion.
Health conditions that Coffee fruit may help support.
Coffee fruit is exceptionally rich in chlorogenic acids and other polyphenols that exert measurable antioxidant activity. Human clinical studies with coffee cherry extract (CCE) have demonstrated reductions in reactive oxygen species (ROS) in blood. In vitro work confirms coffee pulp extract preserves glutathione, superoxide dismutase, and catalase activity in stressed cells.
Multiple clinical trials with CGA-rich coffee extracts have demonstrated significant reductions in systolic blood pressure. A Frontiers in Nutrition RCT (2023) found green coffee extract reduced systolic BP by −5.56 mmHg versus −0.90 mmHg in placebo (p=0.01). Chlorogenic acid improves vasoreactivity and endothelial nitric oxide signaling, which are the proposed mechanisms.
Chlorogenic acid, the primary bioactive in coffee fruit, inhibits glucose-6-phosphatase and modulates glucose absorption, reducing postprandial glucose. A meta-analysis of six RCTs found green coffee extract (sharing coffee fruit's key CGAs) significantly reduced fasting blood glucose (SMD −0.32). An RCT in metabolic syndrome patients found coffee extract significantly attenuated fasting blood sugar versus placebo.
Coffee cherry extract has been shown to reduce reaction time and improve accuracy in cognitively demanding tasks in older adults with mild cognitive decline. Coffeeberry extract also attenuates self-reported mental fatigue during fatiguing cognitive tasks. BDNF upregulation and reduced neuroinflammation are the proposed mechanisms for clearing cognitive fog.
Coffee fruit extract contains chlorogenic acid, epicatechin, and catechin that suppress pro-inflammatory cytokines including TNF-α, IL-6, IL-1β, COX-2, and iNOS. In vitro evidence from macrophage cell models is robust. A 10-week human RCT in type 2 diabetic patients showed green coffee extract (which shares the same key CGA constituents) significantly reduced hs-CRP compared to placebo.
Coffee cherry extract has been specifically studied in older adults with mild cognitive decline in published RCTs. A 28-day RCT in 71 older adults with mild cognitive decline found CCE improved reaction time from the first week, persisting throughout the study. A neurophysiological pilot RCT in older adults with subjective cognitive impairment found CCE produced measurable neurofunctional changes in brain structures supporting cognition.
Human RCTs with whole coffee cherry extract show improvements in working memory accuracy, reaction time, and response inhibition. A 2024 remote RCT (Nutrients) found 200 mg/day CCE acutely reduced omissions and improved accuracy on working memory and inhibitory control tasks versus placebo. A separate 42-day RCT with a whole coffee cherry/phosphatidylserine formula improved working memory accuracy and focus/concentration metrics.
Coffee fruit's polyphenols combat multiple hallmarks of aging: oxidative stress, chronic low-grade inflammation, declining BDNF, and cellular senescence. Human studies confirm BDNF elevation, antioxidant defense, reduced hs-CRP, and cognitive protection in aging populations. The combination of antineuroinflammatory, antioxidant, and neurotrophic mechanisms positions coffee fruit as a multi-target healthy aging ingredient.
Chlorogenic acid-rich coffee extracts have been studied in multiple human RCTs for weight management. A 22-week crossover RCT in 16 overweight adults found green coffee extract reduced body weight by a mean of ~8 lbs with >4% body fat reduction. A separate 12-week double-blind study found 5.4 kg weight reduction with CGA-standardized extract versus placebo.
Chlorogenic acid in coffee fruit has been proposed to improve insulin resistance via hepatic glucose-6-phosphatase inhibition and reduced adipogenesis. Human RCT data with CGA-rich green coffee extracts show directional improvements in HOMA-IR and insulin sensitivity markers in overweight and metabolic syndrome populations, though not all trials show significant effects on insulin levels.
Multiple RCTs with whole coffee cherry extract demonstrate measurable effects on working memory. A 2024 Nutrients RCT found CCE produced an 18.9% improvement in correct working memory responses versus 3.0% in placebo. A Robinson et al. (2019/2020) RCT in older adults with mild cognitive decline showed positive trends in memory testing over 28 days. BDNF elevation by coffee fruit extract may underpin long-term memory-supportive effects.
Polyphenol-rich coffeeberry extract has been shown in published studies to reduce self-reported mental fatigue and increase alertness during cognitively demanding tasks. One published study found low and moderate doses of a caffeine-free coffeeberry extract significantly attenuated increases in self-reported fatigue after fatiguing cognitive tasks. Coffee fruit achieves this without relying on caffeine.
An RCT in metabolic syndrome patients (British Journal of Nutrition, 2018) found green coffee extract significantly attenuated fasting blood sugar versus placebo. Coffee polyphenols are inversely associated with metabolic syndrome risk in epidemiological data. CGA addresses multiple components of metabolic syndrome: blood pressure, triglycerides, blood sugar, and weight.
Coffee polyphenols, especially those found in coffee fruit, enhance energy metabolism and reduce lipogenesis. Chlorogenic acid has been shown to activate fat metabolism in the liver, reduce hepatic triglycerides, and modulate adipogenesis in cell and animal models. Human trials with CGA-rich extracts show effects on body fat, lipid metabolism, and glucose regulation consistent with metabolic enhancement.
Coffee fruit extract robustly elevates BDNF, the primary driver of neuroplasticity via synaptic strengthening, neurogenesis, and neuron survival. A 2013 RCT (British Journal of Nutrition) found 100 mg whole coffee fruit concentrate increased plasma BDNF by 143% at 60 minutes. BDNF is well established to regulate synaptic plasticity and support learning-related neural adaptations.
Coffee berry extract (CBE) in vitro inhibits collagenase (the enzyme that degrades skin collagen) with the highest anti-collagenase activity among tested coffee compounds. A nanoliposome-formulated coffee berry extract was tested in healthy human skin in vivo, assessing skin elasticity and brightness. Coffee cherry pulp extract also suppresses PAH-induced inflammatory aging pathways relevant to skin.
Coffee berry extract demonstrates the highest anti-collagenase activity among coffee-derived compounds in human dermal fibroblast assays, protecting collagen from enzymatic degradation. An in vivo human skin study with nanoliposomal coffee berry extract assessed skin elasticity. Chlorogenic acid in coffee fruit also reduces nitric oxide and oxidative stress in dermal cells, supporting collagen integrity.
RCT evidence with CGA-rich green coffee extracts shows significant reductions in triglycerides. A Frontiers in Nutrition RCT (2023) in type 2 diabetics found GCE reduced triglyceride levels (−49.7 vs. −4.40 mg/dL, p=0.02) and improved TG/HDL ratio versus placebo. Chlorogenic acid retards intestinal fat absorption and reduces hepatic triglyceride synthesis.
Body systems that Coffee fruit may help support.
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